Surgical Management of Type 1 Atresia Ani in Newborn Lamb

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International Journal of Research and Scientific Innovation (IJRSI) | Volume VI, Issue IX, September 2019 | ISSN 2321–2705

Surgical Management of Type 1 Atresia Ani in Newborn Lamb

H.A Bodinga1*, Nura Abubakar1, Shehu Sidi2, A.S Yakubu1, Adamu Abdul Abubakar1

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1Department of Veterinary Surgery & Radiology, Usmanu Danfodiyo University, Sokoto, Nigeria
2Department of Theriogenology & Animal Production, Usmanu Danfodiyo University, Sokoto, Nigeria

Abstract:- A one week old male oudah lamb was presented at the large animal unit of Veterinary Teaching Hospital, Usmanu Danfodiyo University; Sokoto with complaints of distended abdomen and in ability to defecate. Type1 Atresia ani was diagnosed on physical examination. Emergency surgery was carried out following local anaesthesia at the perennial region. Blind rectal pouch was incised and sutured to the skin (rectopexy) using nylon size 2/0. Postoperative antibiotic and analgesic were administered intramuscularly for 5 days. The sutures were removed 2 weeks post-operative. The lamb recovered fully without any complication. Surgical reconstruction is the only course of action for congenital atresia ani in newborn animals.

Keywords: atresia ani, lamb, congenital defect

I. INTRODUTION

Atresia ani (imperforate anus) is a congenital abnormality characterized by persistence of the anal membrane resulting in a thin membrane covering the normal anal canal [2].The condition is characterized by clinical signs of straining, tenesmus, colic, depression and anorexia with abdominal distention [12]. The cause of congenital defects, abnormalities of structure or function present at birth, may be due to genetic or environmental factors, or a combination of both; but the exact cause is poorly understood [10].
There are four major types of intestinal atresia. Type I atresia is a mucosal blockage within the intestinal lumen. In animals with type II atresia, the proximal segment of intestine terminates in a blind end and the distal segment beings similarly with two ends being joined by a fibrous cord devoid of lumen. Type IIIa atresia is similar to type II except that the proximal and the distal intestinal segments blind ends are completely separated and there is a mesenteric defect corresponding to the missing segment of intestine. Animals with type IIIb atresia have a coiled distal segment of intestine. Type IV atresia involves multiple sites of atresia [3],[7].